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Ts1Cje mouse model for Down syndrome research

Intellectual disabilities, hypotonia and cranio-facial dysmorphism are the cardinal characteristics of Down syndrome (DS) individuals. To varying extent, DS individuals also exhibit other developmental problems such as heart defects, vision impairments, hearing loss, hypothyroidism, dental problems...

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Bibliografske podrobnosti
Glavni avtor:	Ling, King Hwa
Format:	Conference or Workshop Item
Jezik:	English
Izdano:	2015
Online dostop:	http://psasir.upm.edu.my/id/eprint/75610/1/Ts1Cje%20mouse%20model%20for%20Down%20syndrome%20research.pdf
Oznake:	Označite Brez oznak, prvi označite!

Podobne knjige/članki

Transcriptional profiling of the postnatal brain of the Ts1Cje mouse model of Down syndrome
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The role of JAK-STAT signalling in the developing brain of Ts1Cje mouse model for Down syndrome
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Expression profiling of genes involved in the development and function of skeletal muscles in Ts1Cje mouse model of down syndrome
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Gene network disruptions and neurogenesis defects in the adult Ts1Cje mouse model of Down syndrome
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Histological and molecular characterisation of hypotonia in adult Ts1Cje mouse model for down syndrome /
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Histological and molecular characterisation of hypotonia in adult Ts1Cje mouse model for down syndrome
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Expression of gamma secretase and notch signallingpathway in TS1CJE mouse model of down syndrome /
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Histological and molecular characterisation of hypotonia in adult Ts1Cje mouse model for down syndrome
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Overexpression of interferon alpha or beta receptors in the brain of adult Ts1Cje mouse model of Down syndrome
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The expression profile of miR-3099 during neural development of Ts1Cje mouse model of down syndrome
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Perturbed metabolic profiles associated with muscle weakness seen in adult Ts1Cje mouse model of Down syndrome
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Expression of gamma secretase and notch signalling pathway in TS1CJE mouse model of down syndrome
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Targeted differential gene expression profiling of skeletal muscles isolated from Ts1Cje mouse model for Down syndrome
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Ts1Cje mouse model for Down syndrome exhibits motor function deficit; the potential role of MRFS and peripheral nervous system
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Expression profiling of notch signalling pathway and gamma-secretase activity in the brain of Ts1Cje mouse model of down syndrome
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Disrupted interferon-related molecular networks and the over-expressed Ifnar1 in the brain of adult Ts1Cje mouse model of Down syndrome
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Transcriptomic and protein expression analyses of skeletal muscles isolated from Ts1cje mouse model for Down syndrome /
od: Leong, Melody Pui Yee.
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Identification of disrupted molecular networks involved in brain maturation and function in the TS1CJE mouse model of down syndrome /
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Transcriptomic and protein expression analyses of skeletal muscles isolated from Ts1cje mouse model for Down syndrome
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Identification of disrupted molecular networks involved in brain maturation and function in the TS1CJE mouse model of down syndrome
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Metabolic profiling of neurospheres derived from embryonic cerebral cortex Ts1Cje mouse model for down syndrome
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Metabolic profiling of neurospheres derived from embryonic cerebral cortex Ts1Cje mouse model for down syndrome /
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The identification of disrupted molecular networks involved in brain maturation and function in the Ts1Cje mouse model of down syndrome.
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Transcriptomic and protein expression analyses of skeletal muscles isolated from Ts1cje mouse model for Down syndrome
od: Leong, Melody Pui Yee
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Identification of disrupted molecular networks involved in brain maturation and function in the TS1CJE mouse model of down syndrome
od: Tan, Kai Leng
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Metabolic profiling of neurospheres derived from embryonic cerebral cortex Ts1Cje mouse model for down syndrome
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Gene and protein expression profiles of JAK-STAT signalling pathway in the developing brain of the Ts1Cje down syndrome mouse model
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Metabolic and functional characterisation of adult skeletal muscle in down syndrome mouse model (Ts1CJe) for insights into hypotonia in human condition /
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Metabolic and functional characterisation of adult skeletal muscle in down syndrome mouse model (Ts1CJe) for insights into hypotonia in human condition
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Metabolic and functional characterisation of adult skeletal muscle in down syndrome mouse model (Ts1CJe) for insights into hypotonia in human condition
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Defects in nerve conduction velocity and different muscle fibre-type specificity contribute to muscle weakness in Ts1Cje Down syndrome mouse model
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Functional transcriptome analysis of the postnatal brain of the Ts1Cje mouse model for Down syndrome reveals global disruption of interferon-related molecular networks
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